Drawing on the notion of parallel interference cancellation, this paper formulates a one-shot linear receiver for the uplink of centralized, possibly cloud-based, radio access networks (C-RANs) operating in a cell-free fashion. This receiver exhibits substantial interference rejection abilities, yet it does not involve any matrix inversions; rather, its structure hinges on the pairwise projections of the users' channel vectors. Its performance is markedly superior to that of matched-filter beamforming, while the computational cost is decidedly inferior to that of an MMSE filter, altogether constituting an attractive alternative in terms of performance vs cost. Furthermore, with a proper sparsification of the channel matrix that it estimates and processes, the proposed receiver can be rendered scalable in the sense of its computational cost per access point not growing with size of the network. Uplink power control is also readily accommodated.

This paper tackles the problem of zero-forcing (ZF) precoding for the downlink of centralized radio-access networks operating in a cell-free fashion. While the customary workhorse of ZF precoding is the channel pseudo-inverse, because of the separate power constraint at each participating access point, the pseudo-inverse is not optimum. Rather, it can be improved upon by other inverses that allow conveying stronger signals (or, equivalently, consuming less power) while respecting the same ZF conditions. Motivated by the enormous disparity in computational cost between simple pseudo-inversion and general inversion, we ascertain the performance disadvantage of the former in a wide range of conditions. The welcome conclusion is that pseudo-inversion is close-to-optimum for all operational regimes of interest.

In this paper, we consider target detection in suspicious tissue via diffusive molecular communications (MCs). If a target is present, it continuously and with a constant rate secretes molecules of a specific type, so-called biomarkers, into the medium, which are symptomatic for the presence of the target. Detection of these biomarkers is challenging since due to the diffusion and degradation, the biomarkers are only detectable in the vicinity of the target. In addition, the exact location of the target within the tissue is not known. In this paper, we propose to distribute several reactive nanosensors (NSs) across the tissue such that at least some of them are expected to come in contact with biomarkers, which cause them to become activated. Upon activation, an NS releases a certain number of molecules of a secondary type into the medium to alert a fusion center (FC), where the final decision regarding the presence of the target is made. In particular, we consider a composite hypothesis testing framework where it is assumed that the location of the target and the biomarker secretion rate are unknown, whereas the locations of the NSs are known. We derive the uniformly most powerful (UMP) test for the detection at the NSs. For the final decision at the FC, we show that the UMP test does not exist. Hence, we derive a genie-aided detector as an upper bound on performance. We then propose two sub-optimal detectors and evaluate their performance via simulations.

While ISI is a common issue in classical communications, it is more challenging and prominent in the context of molecular communication, because one cannot readily combat ISI with classical channel equalization techniques. This is due to the fact that transmitter can only release a positive amount of concentration of a specific molecule into the medium. Previous works have proposed use of chemical reactions to remove molecules from the environment, and to effectively simulate negative signals. However, the differential equation describing a diffusion-reaction process is non-linear. This precludes the possibility of using Fourier transform tools. In this paper, a solution for simulating negative signals based on the diffusion-reaction channel model is proposed. While the proposed solution does not exploit the full degrees of freedom available for signaling in a diffusion-reaction process, but its end-to-end system is a linear channel and amenable to Fourier transform analysis. Based on our solution, a modulation scheme and a precoder are introduced and shown to have a significant reduction in error probability compared with previous modulation schemes, such as concentration shift keying (CSK), pre-equalization, depleted-molecule shift keying (D-MoSK), and molecular concentration shift keying (MCSK). The effects of various imperfections (such as quantization error) on the communication system performance are studied.

In this paper, a diffusion-based molecular communication channel between two nano-machines is considered. The effect of the amount of memory on performance is characterized, and a simple memory-limited decoder is proposed; its performance is shown to be close to that of the best possible decoder (without any restrictions on the computational complexity or its functional form), using genie-aided upper bounds. This effect is adapted to the case of Molecular Concentration Shift Keying; it is shown that a four-bit memory achieves nearly the same performance as infinite memory for all of the examples considered. A general class of threshold decoders is considered and shown to be suboptimal for a Poisson channel with memory, unless the SNR is higher than a computed threshold. During each symbol duration (symbol period), the probability that a released molecule hits the receiver changes over the duration of the period; thus, we also consider a receiver that samples at a rate higher than the transmission rate (a multi-read system). A multi-read system improves performance. The associated decision rule for this system is shown to be a weighted sum of the samples during each symbol interval. The performance of the system is analyzed using the saddle point approximation. The best performance gains are achieved for an oversampling factor of three for the examples considered.