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Ruolin Meng

dblp:197/7638 · DBLP profile ↗
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1ranked-venue papers
1as first author
1since 2021 · last 2025
—ORCID · none

Domains — the database's venue-derived domains; a paper can count in several

Artificial intelligence and machine learning · 1 · 1 first-author · 1 since 2021

Expertise — from the expertise taxonomy: the topics of the expert's papers under the CCF categories. A weight counts papers with recency: 1 for a paper about the topic, 0.3 when the topic is its context, halved every five years.

Artificial intelligence
1 paper
Probabilistic and Bayesian machine learning · 91% Deep learning architectures and training · 9%

Topics — the 4 heaviest of 4, each with the papers that count most for it

TopicWeightPapersLastEvidence papers
Machine learning › Probabilistic and Bayesian machine learning
causal inference
0.912025
Coupling Generative Modeling and an Autoencoder with the Causal Bridge · NeurIPS 2025
Machine learning › Probabilistic and Bayesian machine learning › causal inference
latent confounders
0.912025
Coupling Generative Modeling and an Autoencoder with the Causal Bridge · NeurIPS 2025
Machine learning › Probabilistic and Bayesian machine learning › causal inference › causal effect estimation
treatment effect estimation
0.912025
Coupling Generative Modeling and an Autoencoder with the Causal Bridge · NeurIPS 2025
Machine learning › Deep learning architectures and training
autoencoder
0.312025
Coupling Generative Modeling and an Autoencoder with the Causal Bridge · NeurIPS 2025

Methods — techniques the papers use, named apart from their topics

generative modeling · 0.9autoencoder · 0.9
YearPublicationVenuePosition
2025 Coupling Generative Modeling and an Autoencoder with the Causal Bridge
abstract
We consider inferring the causal effect of a treatment (intervention) on an outcome of interest in situations where there is potentially an unobserved confounder influencing both the treatment and the outcome. This is achievable by assuming access to two separate sets of control (proxy) measurements associated with treatment and outcomes, which are used to estimate treatment effects through a function termed the *causal bridge* (CB). We present a new theoretical perspective, associated assumptions for when estimating treatment effects with the CB is feasible, and a bound on the average error of the treatment effect when the CB assumptions are violated. From this new perspective, we then demonstrate how coupling the CB with an autoencoder architecture allows for the sharing of statistical strength between observed quantities (proxies, treatment, and outcomes), thus improving the quality of the CB estimates. Experiments on synthetic and real-world data demonstrate the effectiveness of the proposed approach relative to state-of-the-art methodology for causal inference with proxy measurements.
Ruolin Meng, Ming-Yu Chung, Dhanajit Brahma, Ricardo Henao, Lawrence Carin
NeurIPS1