Reflectance acquisition from sparse images has been a long-standing problem in computer graphics. Previous works have addressed this by introducing either material-related priors or illumination multiplexing with a general sampling strategy. However, fixed lighting patterns in multiplexing can lead to redundant sampling and entangled observations, making it necessary to adaptively capture salient reflectance responses in each shot based on material behavior. In this paper, we propose combining adaptive sampling with illumination multiplexing for SVBRDF reconstruction from sparse images lit by a planar light source. Central to our method is the modeling of a sampling importance distribution on lighting surface, guided by the statistical nature of microfacet theory. Based on this sampling structure, our framework jointly trains networks to learn an adaptive sampling strategy in the lighting domain, and furthermore, approximately separates pure specular-related information from observations to reduce ambiguities in reconstruction. We validate our approach through experiments and comparisons with previous works on both synthetic and real materials.

The arterial aneurysm refers to localized dilation of blood vessel wall and is common in general population. The majority of aneurysm cases remains asymptomatic until a sudden rupture which is usually fatal and of extremely high mortality (~ 50-60%). Therefore, early diagnosis, prevention and management of aneurysm are in urgent need. Unfortunately, current understanding of disease driver genes of various aneurysm subtypes is still limited, and without appropriate biomarkers and drug targets no specialized drug has been developed for aneurysm treatment. In this research, aneurysm subtypes were analyzed based on protein-protein interaction network to better understand aneurysm pathogenesis. By measuring network-based proximity of aneurysm subtypes, we identified a relevant closest relationship between aortic aneurysm and aortic dissection. An improved random walk method was performed to prioritize candidate driver genes of each aneurysm subtype. Thereafter, transcriptomes of 6 human aneurysm subtypes were collected and differential expression genes were identified to further filter potential driver genes. Functional enrichment of above driver genes indicated a general role of ubiquitination and programmed cell death in aneurysm pathogenesis. Especially, we further observed participation of BCL-2-mediated apoptosis pathway and caspase-1 related pyroptosis in the development of cerebral aneurysm and aneurysmal subarachnoid hemorrhage in corresponding transcriptomes.